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题名

Absence of GdX/UBL4A Protects against Inflammatory Diseases by Regulating NF-kappa B Signaling in Macrophages and Dendritic Cells

作者
通讯作者Wu, Li; Chang, Zhijie
发表日期
2019
DOI
发表期刊
ISSN
1838-7640
卷号9期号:5页码:1369-1384
摘要
Nuclear factor-kappa B (NF-kappa B) activation is critical for innate immune responses. However, cellular-intrinsic regulation of NF-kappa B activity during inflammatory diseases remains incompletely understood. Ubiquitin-like protein 4A (UBL4A, GdX) is a small adaptor protein involved in protein folding, biogenesis and transcription. Yet, whether GdX has a role during innate immune response is largely unknown.
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语种
英语
学校署名
其他
资助项目
Tsinghua Science Foundation[20121080018] ; Tsinghua Science Foundation[20111080963]
WOS研究方向
Research & Experimental Medicine
WOS类目
Medicine, Research & Experimental
WOS记录号
WOS:000459354100012
出版者
来源库
Web of Science
引用统计
被引频次[WOS]:20
成果类型期刊论文
条目标识符http://sustech.caswiz.com/handle/2SGJ60CL/26705
专题南方科技大学医学院_生物化学系
生命科学学院_生物系
作者单位
1.Tsinghua Univ, Sch Med, State Key Lab Membrane Biol, Beijing 100084, Peoples R China
2.Tsinghua Univ, Inst Immunol, Sch Med, Beijing 100084, Peoples R China
3.Third Mil Med Univ, Inst Immunol, PLA, Chongqing 400038, Peoples R China
4.Shanghai Jiao Tong Univ, Chinese Acad Sci, Inst Hlth Sci, Shanghai Inst Biol Sci,Sch Med, Shanghai 200025, Peoples R China
5.South Univ Sci & Technol China, Dept Biochem, Shenzhen 518055, Peoples R China
推荐引用方式
GB/T 7714
Liu, Chunxiao,Zhou, Yifan,Li, Mengdi,et al. Absence of GdX/UBL4A Protects against Inflammatory Diseases by Regulating NF-kappa B Signaling in Macrophages and Dendritic Cells[J]. Theranostics,2019,9(5):1369-1384.
APA
Liu, Chunxiao.,Zhou, Yifan.,Li, Mengdi.,Wang, Ying.,Yang, Liu.,...&Chang, Zhijie.(2019).Absence of GdX/UBL4A Protects against Inflammatory Diseases by Regulating NF-kappa B Signaling in Macrophages and Dendritic Cells.Theranostics,9(5),1369-1384.
MLA
Liu, Chunxiao,et al."Absence of GdX/UBL4A Protects against Inflammatory Diseases by Regulating NF-kappa B Signaling in Macrophages and Dendritic Cells".Theranostics 9.5(2019):1369-1384.
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